ACUTE CORONARY SYNDROME AS A CAUSE OR A CONSEQUENCE OF A SEVERE THORACIC BLUNT TRAUMA – A CASE REPORT
Abstract
Background: Acute coronary syndrome associated with severe thoracic trauma may be a concomitance, a coincidence or a subsequent event, requiring specific adaptation of emergency management. The purpose of the study- to highlight the special circumstances of an ACS occurrence in a severe thoracic blunt trauma
Material and method: Clinical case presentation - A 29 years old patient with severe thoracic blunt trauma, secondary brought to a level I trauma center ED. First assessment - serious chest trauma, malignant cardiac dysrhythmias, severe head trauma, spinal cord and pelvic fracture, progressive shock. Increased levels of cardiac biomarkers with atypical progression. Several hypotheses for the acute cardiac damage etiology considered, as toxic, ACS both causing or following the accident, but, taking into account the kinematics and severity of thoracic trauma, it was also considered the possibility of cardiac contusion occurred. Considering circumstances, the management has been orientated to life-threatening lesions and damage control and advance imagistic to confirm cardiac injury causes.
Conclusions: The strong suspicion for ACS associated with significant thoracic blunt trauma poses questions about his etiology (coronary artery disease, aortic or coronary artery traumatic disrupture), and, subsequently, about possible primary reperfusion procedures required. The concomitance of brain injury, hemorrhage and pulmonary contusion limited treatment of a potential ACS from CAD, fast volume replacement and ventilation strategies, in the context of shock and risk of secondary brain and spinal cord injury. HEMS has to be involved with a primary mission to evacuate directly to a level I trauma center this type of patients.
Downloads
References
2. Bernard SA, Gray TW, Buist MD, et al. Treatment of comatose survivors of outof-hospital cardiac arrest with induced hypothermia. N Engl J Med 2002;346:557-63
3. Buduhan G., McRitchie D. I.. Missed Injuries in Patients with Multiple Trauma. The Journal of Trauma, Injury, Infection, and Critical Care, 2000 Oct;49 (4): 600 - 5
4. Bulger EM, Guffey D, Guyette FX, MacDonald RD, Brasel K, Kerby JD, Minei JP, Warden C, Rizoli S, Morrison LJ, Nichol G. Resuscitation Outcomes Consortium Investigators. The impact of prehospital mode of transport after severe injury: a multicenter evaluation of the Resuscitation Outcomes Consortium. J Trauma Acute Care Surg. 2012 Mar;72(3):567-73; discussion 573-5; quiz 803. doi:10.1097/TA.0b013e31824baddf.
5. Hachimi-Idrissi S, Corne L, Ebinger G, Michotte Y, Huyghens L. Mild hypothermia induced by a helmet device: a clinical feasibility study. Resuscitation 2001; 51: 275 – 81.
6. Penela D, Magaldi M, Fontanals J, Martin V, Regueiro A, Ortiz JT, Bosch X, Sabaté M, Heras M. Hypothermia in acute coronary syndrome: brain salvage versus stent thrombosis? J Am Col Cardiology 2013; 61(6):686-7.
7. Pryor J.P., Pryor R.J., Stafford P. Initial phase of trauma management and fluidDownloaded from Medico Research Chronicles“Acute coronary syndrome as a cause or a consequence of a severe thoracic blunt trauma – A case report” Rotaru L. T., et al., Med. Res. Chron., 2017, 4 (2), 169-174 Medico Research Chronicles, 2017 174 resuscitation. Trauma Reports, 2002, 3(3): 1-12
8. Talving P, Teixeira PG, Barmparas G, DuBose J, Inaba K, Lam L, Demetriades D - Helicopter evacuation of trauma victims in Los Angeles: does it improve survival? World J Surg. 2009 Nov;33(11):2469-76. doi: 10.1007/s00268-009-0185-1
9. The hypothermia after cardiac arrest study group. Mild therapeutic hypothermia to improve the neurologic outcome after cardiac arrest. N Engl J Med 2002; 346:549-56.
10. Vinson D.R, Bradbury D.R. Etomidate for procedural sedation in emergency medicine. Ann Emerg Med, 2002, (Jun)39(6): 592-8.
11. Wang H.E., Yealy DM. Out-of-hospital rapid sequence intubation: is this really the "success" we envisioned?. Ann Emerg Med; 2002 (Aug), 40(2):168-71