Anomalies of the genetic complement and its role in C3 Glomerulopathies and development of Hemolithic Uremic Syndrome

  • Juan Esteban Tafur Delgado Second-year Pediatric Resident Of The University Of Sinú
  • Carlos Andrés Pérez Lozano General Medical University Foundation of Health Sciences, Bogotá
  • Lina Marcela Ortiz Roncallo General Medical University Of The Sabana, Bogotá
  • Silvia Juliana Rodríguez Pérez Intern At The University Of Sinú, Montería
  • Amaury Gustavo López Agámez Intern At The University Of Sinú, Montería
  • María José Álvarez Gómez Intern At The University Of Sinú, Montería
  • Ricardo Javier Galván Zúñiga Intern At The University Of Sinú, Montería
  • Ana María Garcés Pérez General Medical Of The University Of Magdalena
Keywords: genetic complement anomalies, glomerulopathy, active C3

Abstract

Let us remember that C3 glomerulopathy is the product of an abnormal activation in the alternative complement pathway, which leads to an excessive production of active C3 and its degradation products

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References

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How to Cite
1.
Delgado JET, Lozano CAP, Roncallo LMO, Pérez SJR, Agámez AGL, Gómez MJ Álvarez, Zúñiga RJG, Pérez AMG. Anomalies of the genetic complement and its role in C3 Glomerulopathies and development of Hemolithic Uremic Syndrome. Med. res. chronicles [Internet]. 2021Jul.20 [cited 2024Dec.22];8(4):303-4. Available from: https://medrech.com/index.php/medrech/article/view/516
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Editorial Note